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Presented for research literature review only Misra S (2024)

Although moderate exposure to fatty acids increased apoB100 secretion, prolonged exposure leads to ER stress and posttranslational degradation of apoB100, and consequently decreased apoB100 secretion, both in vivo and in vitro hereby linking ER stress to NAFLD progression through apoB100 inhibition [114, 115] (Fig

Glucagon-like peptide 1 treatment reverses vascular remodelling by downregulating matrix metalloproteinase 1 expression through inhibition of the ERK1/2/NF-B signalling pathway

This inhibition leads to decreased numbers and reduced migration of CD4 + T cells, as well as a reduction in the release of IL-5 and IL-13 (Toki et al., 2021

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