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10.1016/j.celrep.2018.10.018 112

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C-reactive protein and the prediction of cardiovascular events among those at intermediate risk: moving an inflammatory hypothesis toward consensus

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Collectively, our data suggest that T3 might also be able to increase the production of non-12-OH FXR-antagonistic BA species in human likely consequent to the increased synthesis of non-12-OH BAs, thereby potentiating the GLP-1 and insulin production by suppressing the intestinal FXR signaling (Fig

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