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By conducting in-depth analysis of the diet-microbiota-immunometabolism regulatory network, through targeted dietary modifications to adjust nutrient availability in the TME, reshaping the gut microbiota with probiotics or prebiotics to enhance immunometabolic activity, and combining immune checkpoint inhibitors with metabolism-targeted drugs, it may be possible to disrupt tumor metabolic adaptation and immune escape at a systemic level, thereby achieving personalized and precision therapy

Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo

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Presented for research literature review only Hayes JD et al