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Additionally, the asialoglycoprotein receptor (ASGPR) is highly expressed on the surface of hepatocytes, capturing the terminal galactose or N-acetylgalactosamine residues exposed on glycoproteins to drive RME activity, thereby achieving efficient clearance of ASGPR from the bloodstream

While these treatments are not cures, they represent a hopeful direction: targeting redox balance and inflammation as part of autism management