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SGLT2i were prescribed in a similar manner in the later and earlier periods, although when an SGLT2i was prescribed as an add-on therapy, it was less often added to an ACEi/ARB in the later period than the earlier period (54.0% vs

25,26,27 In contrast, mutations in POLE/POLD1 enhance immunogenicity through hypermutation, 28 while excessive copy number alterations often drive immunosuppressive phenotypes and resistance

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