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Two downstream consequences are mechanistically central: nicotinamide is preserved for re-entry into the NAD+ salvage pathway (raising intracellular NAD+ availability), and SAM is preserved for other methylation reactions
Conclusion Our study reveals that the activation of NLRP3 inflammasome caused by mitophagy dysfunction plays an important role in cardiac hypertrophy induced by pressure overload, and semaglutide ameliorates the cardiac hypertrophy by improving cardiac mitophagy to suppress the activation of NLRP3 inflammasome
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Necrosis (centrilobular, midzonal, periportal) might be accompanied by other histological changes (fatty change, congestion, hemorrhage, inflammation, bile stasis, fibrosis, etc.) to form a myriad of pathological changes
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