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May 2013;57(5):824-832

One proposal is that hypofunction of NMDA receptors (NMDAR) located on inhibitory GABA inter-neurons leads to reduced activity of these inter-neurons, resulting in increased pyramidal glutamatergic neurotransmission and the diverse symptoms occurring in acute psychosis [3]

The alteration of NAD + metabolism in sPD cells contributes to the activation of SIRT2 and subsequently reduces the acetylation level of -tubulin

Neutrophil gelatinase-associated lipocalin: its response to hypoxia and association with acute Mountain sickness

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