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It is possible that these differences are due to the DPP-4 inhibitors blocking the degradation of peptides such as GIP and neuropeptide Y, which exert opposite effects on gastric motility and the central nervous system (CNS) control of appetite [5]
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Moreover, GLP-1 suppresses appetite by inhibiting AMP-activated protein kinase (AMPK) activity in the ventromedial hypothalamus (VMH) [25, 26]
In addition, GLP-1R is recognized as a crucial regulator of cellular glucose metabolism
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