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The article says both agents influence AMPK signaling and mitochondrial function but through different pathways, which it argues makes a pharmacological interaction unlikely at standard clinical doses

Longer follow-up will tell us whether improvements in frailty lead to fewer hospitalizations and better survival

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may be involved in the penile vascular endothelial dysfunction of MetS-associated ED, and are reflected in the main pathological features of MetS such as glucose metabolism disorder, hypertension, dyslipidemia, and obesity