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Additionally, the translational relevance of these findings likely differs by disease phenotype

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R., Tippmann-Peikert, M., Slocumb, N

[6][7] The four structural substitutions within Modified GRF 1-29 are thought to confer resistance to DPP-IV-mediated cleavage, potentially extending implicationive receptor engagement relative to the unmodified GRF(1-29) sequence while preserving a physiologically plausible pulsatile pharmacokinetic profile. Ipamorelin activates GHS-R1a, a constitutively active GPCR expressed on pituitary somatotroph cells and in hypothalamic nuclei

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