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In addition, several sources of Se including Na 2 SeO 3 , Sel-Met, and selenite containing Se compounds all showed positive effects on cell viability in various cells including human immature dendritic cells, human lens epithelial cells (44), chondrocyte ATDC5 cells (42), fibroblast HT1080 cells, and primary porcine macrophages cells (45, 46)
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Initially, mechanical forces disrupt glomerular endothelial cells and their protective glycocalyx, followed by activation of podocyte stress responses and mesangial cell proliferation with excessive extracellular matrix deposition (50, 51)
Functional validation using receptor-deficient models confirms that 3-adrenergic signaling is required for these effects, as disruption of this pathway attenuates peptide-induced lipolysis
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