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Also of interest is that GSH supplementation resulted in a reduction of oxidized GSH levels in blood, suggestive of a reduction in levels of oxidative stress
8): an oxidative biotransformation product of BBR generated through gut microbial metabolite, dose-responses attenuation of hepatic steatosis and obesity-related pathophysiological markers in HFD-induced NAFLD rodent models, surpassing BBR at equivalent dosing (100 mg/kg)
Early diagnosis and referral of patients with high-risk lesions to an appropriate specialist service is key to improving outcomes
Similar results were obtained with human HepG2 hepatocyte cell line (Huang et al., 2018)
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