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IGF-1, ,
The rationale behind cyclical dosing connects directly to epitalon's mechanism of action
doi: 10.3389/fphar.2025.1718803 Received 04 October 2025 Revised 07 November 2025 Accepted 10 November 2025 Published 26 November 2025 Volume 16 - 2025 Edited by Giuseppe Remuzzi, Istituto di Ricerche Farmacologiche Mario Negri IRCCS, Italy Reviewed by Muhammad Saboor, University of Sharjah, United Arab Emirates Ferruh Artunc, University of Tbingen, Germany Updates Copyright 2025 Deng, Gong, Huang, Zhou and Ren
| Wadivkar P, Jebasingh F, Thomas N, Yajnik CS, Vaag AA, Kibirige D, Bahendeka S, Bajaj M, Boyne MS, Bavuma C, Abodo J, Wild S, Kolthur-Seetharam U, Mukhopadhyay S, Somasundaram NP, Schwarz P, Christensen DL, Pathan F, Jones AG, Mathur S, Kehlenbrink S, Hawkins M
This truncated soluble receptor inhibited the autophosphorylation of the endogenous IGF-1R as well as induced extensive apoptosis in vivo and inhibited tumorigenesis in syngeneic rats
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