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Genetic deletion of the Fmr1 gene leads to a depletion of fragile X mental retardation protein (FMRP), resulting in the activation of the metabotropic glutamate receptors mGluR [331, 332]
Grams, M
But it aligns with the general principle that slower escalation produces fewer side effects across all categories
On the other hand, mitochondria can effectively reduce ETC-derived ROS by activating uncoupling proteins, which lower the inner membrane potential a key driver of ROS production, thereby preserving cellular redox homeostasis [142]
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