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BPC-157 and TB-500 also reduce inflammation in animal studies, but as a secondary effect of their tissue-repair pathways, not as a primary mechanism
A crucial next step in drug development, several experts told Undark, is to test for safety and efficacy in humans
TB-500 is a 43-residue, 4963.4 g/mol thymosin beta 4 sequence acting through G-actin sequestration and integrin-linked kinase signalling, studied mainly in cardiac, corneal, dermal and skeletal muscle models
Does BPC-157 go bad
The tissue lacks the robust blood supply that muscles enjoy, receives constant mechanical loading that disrupts healing, and often develops fibrotic scar tissue rather than healthy organized collagen
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