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Several weight-independent mechanisms may contribute to the cardioprotective effects of GLP-1RAs, including direct vascular and myocardial effects [29], improved plaque stability [29], anti-inflammatory benefits [30], and modulation of prothrombotic [31] and neurohormonal activity [15]

This may indicate de novo L-carnitine formation from endogenous TML

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Determination of the receptorligand co-crystal structures and mutational studies have led to a two-step model of peptide binding and receptor activation 17,18,19,20

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