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The absorption at 290 nm was carried out using the clear supernatant fraction obtained
In conclusion, the current study suggests that loss of hepatic GLP-1R in NAFLD patients is responsible for low responsiveness to GLP-1 therapy
Furthermore, it suppressed RAW264.7 cell viability and nuclear translocation of NF-B p65, suggesting its anti-inflammatory mechanism may be related to the NF-B signaling pathway
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