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These results have raised the question of whether benzodiazepines can cause structural brain damage

metabolite Ac-LKKTE identified as potentially more active than parent compound Paradoxical finding: Both peptides show biological effects persisting hours to days after administration despite rapid plasma clearance Possible explanations: Tissue retention, active metabolites, or persistent signaling cascade activation Excretion Pathways Limited data exists on excretion for both peptides[22]: Likely renal elimination of peptide fragments Hepatic metabolism may contribute to clearance No accumulation detected in chronic dosing studies (animal models) Combined excretion kinetics have not been characterized in any species The disconnect between short plasma half-lives and prolonged biological effects represents a key area requiring mechanistic clarification for both individual peptides and their combination

Oxidative and inflammatory processes may affect a plaques lipid content, immune-cell activity, fibrous covering, and vulnerability to rupture

Still, they are examined specifically for attention, sustained concentration, and resistance to mental fatigue rather than for long-term neuroprotection

doi: 10.1038/s41593-019-0393-4 32 DavuluriGVNChanCH