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134 YueP.ZhouZ.KhuriF
[57] It does this by upregulating phosphorylated IRS-1 and activating pathways (like AMPK/SIRT1) that promote the transport of the GLUT4 glucose transporter to cell membranes in muscle and adipose tissue, thereby increasing overall cellular glucose uptake
C.VousdenK
Impaired mitophagy permits accumulation of dysfunctional mitochondria, establishing a feed-forward cycle of oxidative stress within the supraspinatus
The BSH recommends that treatment decisions should be based on clinical presentation rather than borderline laboratory values alone
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