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Underlying Mechanisms: The exact triggers for the development of Hashimotos remain complex and multifaceted

In vitro, TUG469 reduced 6-OHDA (6-hydroxydopamine)-induced neurotoxicity, whereas in a 6-OHDA-induced mouse model of PD, TUG469 improved motor deficits, preserved both dopaminergic fibres in the striatum and SN and reduced microgliosis and astrogliosis [66]

This matters because combination therapy is not a short-term strategy

* P 0.05, ** P 0.01, *** P 0.001, and **** P 0.0001 by unpaired t test ( A ), or 2-way ANOVA followed by Tukey post hoc tests ( C O )

6-phosphogluconolactone is then converted to 6-phosphogluconate via the lactonase enzyme