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It reveals that altered glutathione and AA homeostasis induced by Trkb deletion in iSPNs is directly linked to early dopaminergic dysfunction and disrupted energy metabolism followed by age-dependent motor impairment and strongly supports the relevance of our model to elucidate the mechanisms underlying the vulnerability of iSPNs in neurodegenerative conditions
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After being returned to their racks mice were checked for signs of pain and distress and their weight monitored daily
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