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Functional studies in receptor-deficient models further confirm that 3-adrenergic signaling is essential to its activity, positioning AOD 9604 as a tool for dissecting receptor-mediated lipid mobilization [1]
The peptide may potentially trigger GHRH receptors GRF (1-29)
The effects of human GH and its lipolytic fragment (AOD 9604) on lipid metabolism following chronic treatment in obese mice and beta(3)-AR knock-out mice