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Heat stress can preferentially induce germ-cell injury through ROS accumulation, DNA damage, mitochondrial dysfunction, and activation of caspase-dependent apoptotic pathways (Robinson et al., 2023)
And so I don't want to get into the whole beige fat, brown
GH, epidermal growth factor, basic fibroblast growth factor)[7072], nutrition[73, 74], and injury (hypoxic/ischemic, stereotactic, electrolytic and cryogenic injuries, as well as induced demyelination and experimental autoimmune encephalomyelitis)[7581], influence its in vivo brain expression
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