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Furthermore, in most studies concerning the chronic effects of GLP-1 on BP, BP is not the primary or pre-established endpoint and most data comes from studies in T2DM

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5,21,22 Although causality is often difficult to establish, clinical case reports and postmarketing pharmacovigilance studies suggest a classwide signal that warrants continued monitoring

It has shown efficacy in animal models of inflammatory bowel disease (IBD), gastric ulcers, and "leaky gut." Systemic anti-inflammatory effects: It appears to dampen inflammatory cascades without the immune-suppressing risks associated with corticosteroids