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This supports immune function without relying only on antioxidants

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Critically, the excessive buildup of iron within the brain establishes a self-sustaining cycle: iron accumulation promotes -amyloid (A) aggregation and tau hyperphosphorylation (Tran et al., 2022), while A plaques in turn sequester iron via their metal-binding domains, increasing local Fenton reaction activity (Tang et al., 2024)

This precision approach helps move beyond one-size-fits-all protocols toward individualized treatment planning

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