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Experimental findings suggest that it supports muscle repair by improving local vascular responses at sites of injury
In smoking-associated chronic inflammatory environments, the upregulation of PTPRS and SFRP2 on fibroblasts aligns with previous studies demonstrating increased expression of these markers in fibroblasts with a pro-inflammatory phenotype
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Turbulent flow promotes NF-B and AP-1 activity that upregulate pro-inflammatory and pro-atherogenic genes such as ICAM-1, E-selectin, platelet-derived growth factor (PDGF)-BB, IL-1a, bone morphogenic protein-4 (BMP-4), monocyte chemotactic protein-1 (MCP-1), and ET-1 [211]
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