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Concurrently, XO contributes to the production of reactive oxygen species (ROS)
Thus, in alignment with the interpretation from Thomas et al, perhaps changes in cerebrovascular resistance observed in RT are to be separated from vascular pathology and recontextualized as a favourable adaptation, as increased resistance may suppress transmission of pulsatile blood flow into the cerebral circulation during- and post-resistance exercise (Koch et al., 2005)
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