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In models of nonalcoholic steatohepatitis (NASH), GLP-1 analogs have been shown to attenuate hepatic inflammation by reducing M1 macrophage infiltration and inhibiting NF-B activity, thereby lowering the expression of pro-inflammatory mediators such as IL-6, TNF-, and iNOS (Yusta et al., 2015)
Studies have shown that bitter-tasting pharmaceuticals and substances, including polyphenols such as isosinensetin, bind to bitter taste receptors, and unlike other TAS2R agonists, stimulate GLP-1 secretion in NCI-H716 cells [192] (Fig
Kisspeptin-10: Best Emerging Research Peptide Kisspeptin-10 ranks third because it sits even further upstream, helping stimulate GnRH and then LH pulse activity
