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Since inhibition of FXR in intestinal L cells is able to promote GLP-1 release 11 , changes in BA composition or profiles are likely to affect glucose homeostasis by controlling insulin secretion via modulating GLP-1 production
This activation has been shown to upregulate genes involved in collagen synthesis, cell proliferation, and differentiation while potentially downregulating matrix metalloproteinases
Peak metabolic effects occur at month 3 and beyond, when the body fully responds to the medications dual-action mechanism
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