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A more recent randomized, double-blind, placebo-controlled study in 30 patients with type 2 diabetes found that intravenous infusion of 600 mg of lipoic acid improved the response to the endothelium-dependent vasodilator acetylcholine but not to the endothelium-independent vasodilator, glycerol trinitrate (63)

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To validate our findings that liraglutide increases INS and PCSK1 expression in human cells, immunostaining was performed on islets from a human donor (Supplemental Figure 5A) for GLP-1, insulin, and glucagon after 24 hours of treatment with saline or liraglutide

This reflects the difference in the real-world clinical practice where patients who experienced side effects might more easily decide to stop the therapy as they perceived benefits from naltrexone-bupropion outweighed by additional considerations such as cost