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Furthermore, we provide first data that the GSH level in human neurons may be mediated by NMDAR activation since the protective effect of enhanced network activity could be blocked with an NMDAR antagonist APV, consistent with the previous report (Fig
Gregory Westcott, Beth Israel Deaconess Medical Center and Harvard Medical School, United States Updates Copyright 2023 Muller, Stenvers, Malekzadeh, Holleman, Painter and Siegelaar
Abstract Maternal cigarette smoke exposure (SE) causes detrimental changes associated with the development of chronic neurological diseases in the offspring as a result of oxidative mitochondrial damage
Two patients of mine who took extended-release stimulants while on a GLP-1 agonist may have also seen increased development of tolerance to the stimulant effect
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