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This suggestion was further insisted as a potential mechanism of TB-500 by the authors of the research study, who also commented that "transfection of anti-miR-146a nucleotides reversed the inhibitory effect of T4 on IRAK1 and TRAF6." Thus, TB-500 also appears to potentially work for healing by exerting anti-inflammatory action.(6)

So, while the explanation touches on real aspects of neuropathy and B12 deficiency, it might oversimplify or misrepresent some of the complex interactions in the nervous system

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BPC-157 and KPV are both very well tolerated