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This protein complex increases the activity of various inflammatory genes and their products

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We also take 30% of surviving hepatocytes to be the boundary between surviving and dying without NAC rescue [38]

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This mechanism establishes OLA as a promising anti-fibrotic therapeutic candidate ( Mitochondria-targeted antioxidants or CYP2E1 inhibition prevents this toxicity, highlighting CcO dysfunction as a key pathological mechanism ( Daphnia magna through its reactive metabolite NAPQI