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In our experiments, exogenous rapamycin significantly decreased the proportion of SA--Gal-positive cells and p16 expression, whereas autophagy inhibitors 3-MA and Baf-A1 produced opposite effects

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Future research can apply our analytic framework to perform a real-world-study-based CEA for either corroborating our findings or translating more real-world comparative effectiveness research to inform clinical professionals and health policymakers when prioritizing treatment strategies for patients with T2D given limited healthcare resources