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In a model proposed in the literature, a decrease in the level of oxidative stress may contribute to the apoptosis of malignant cells and may serve as one of the mechanisms by which bilirubin screens malignant cells (76) ( Figure 2 )

Such monitoring may include assessment of dietary protein intake, body composition trajectories, and micronutrient status in order to maintain proportionality between oxidative metabolic demand and substrate-supported regenerative capacity This perspective aligns with emerging calls to integrate pharmacologic and nutritional paradigms in obesity treatment, recognizing that durable metabolic adaptation requires coordination between drug-mediated signaling effects and substrate-dependent metabolic processes [103, 115]

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