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Elevated levels of extracellular glutamate in diverse brain trauma contexts inhibited system X c transportation, which in consequence triggered ferroptosis ( c can induce cell death by lowering uptake of cysteine, which leads to GSH depletion and ultimately impairs the cells ability to resist oxidative stress ( GSH depletion following TBI is seen in several experiment cases and usually contributes to secondary injury (Yang et al., 2014

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