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Collectively, these data might indicate that the diminished ROS buffering of GSH-deficient cells leads to an increased activation of the mTOR pathway which could drive to the MZB-like properties of Gclc -deficient FoB
CJC-1295, also referred to as Mod GRF 1-29, has been investigated as an analog of somatocrinin, with potential to stimulate growth hormone-releasing hormone (GHRH) receptors
Eur J Pediatr 157:738742
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