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Monocytes exhibit altered subset composition, impaired phagocytic capacity, and metabolic reprogramming involving mitochondrial and lysosomal dysfunction, partly linked to mutations in the LRRK2 and GBA1 genes, which further sustain inflammation and alpha-synuclein aggregation
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NMR and HPLC profiling of bee pollen products from different countries
Two common LPL gene variants (447 Ter and 291 Ser) were associated with metabolic syndrome through their effect on high TG and low HDL cholesterol [149]
GLP-1 did not increase myocardial oxygen consumption or blood flow in humans or in swine, increasing p38-MAPK activity in lean, but not obese cardiac tissue [48]
