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The treatment of ASD remains challenging due to its etiological heterogeneity and the limited efficacy of current pharmacological interventions (Masi et al
Matsuzaki, K
Third, it upregulates VEGFR2 and activates the VEGFR2-Akt-eNOS pathway, accelerating blood vessel formation and blood flow recovery in ischaemic tissue, a mechanism confirmed in endothelial cell culture and rat hind-limb ischaemia models by Hsieh 2017
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It has been studied for sleep, opioid and alcohol withdrawal, stress, and pain modulation
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