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Even a few hours at room temperature can reduce potency

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These metabolites reduced inflammatory lipid mediator production in human neutrophils and monocytes, with potency comparable to pharmaceutical 5-LO inhibitors (Pein et al., 2018)

Hence, prolonged IGF-1 or IGF-1 gene therapy could disrupt the nuclear p53progerin interaction to repress oxidative SIPS via activating the PI3K/AKT1 pathway and inhibiting p53 nuclear translocation

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