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The most consistent problem was the near-total absence of human clinical trial data for the proposed routes of administration

Blockade of BRD4HiBiT degradation by MLN4924 and carfilzomib was not observed at higher concentrations approaching 10 M, which we suspect is because of the transcriptional consequences of BET bromodomain inhibition, as JQ1 itself decreases BRD4HiBiT signal at these high concentrations (Extended Data Fig

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The result is increased bone formation on both trabecular and cortical bone surfaces