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In addition, as an epigenetic modification, DNA methylation can be involved in disease progression and serve as a marker of environmental exposures, such as tobacco smoking, lipid peroxidation, and inflammation (Gao et al., 2019

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[172] reported that LCMT-1 overexpression protected animals from A-induced impairments, whereas overexpression of phosphatase methylesterase 1 worsened A neurotoxicity [172]

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H 2 O 2 oxidizes cysteine residues (Cys257, Cys268, and Cys568) on RIPK1, a process facilitating RIPK3 recruitment to the necrosome 70