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Its cellular-level mechanism appeals to those wanting to optimize metabolic function rather than override it
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Because AOD9604 does not stimulate IGF-1 production and lacks strong GH receptor agonism, it may present a safer alternative for long-term OA treatment [1]
Helps prevent and treat Vitamin B12 deficiency
[36] I have more information on this in the Advanced Protocols section of my book Hashimotos Protocol
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