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Because flavonoid accumulation is often regulated by transient signaling events and feedback mechanisms, static network models provide only a partial view of pathway behavior

Again, evidence is primarily animal-based, not FDA approved

View Study [6]"Expression and activation of matrix metalloproteinases in wounds: modulation by GHK-Cu" Simeon A, Monier F, Emonard H, Gillery P, et al., 1999 Finding: GHK-Cu was shown to intelligently regulate wound remodeling enzymes (MMPs)increasing them when needed for tissue restructuring and decreasing them to prevent excessive breakdown

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The assay mixture was composed of 250 M phosphoribosyl pyrophosphate (PRPP), 200 M orotate and 5 mM MgCl 2 in 20 mM Tris-HCl pH 8.0 buffer