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Many preclinical studies have indicated that Ea exhibits neuroprotective effects by anti-apoptosis and reduction of neuroinflammation, mediated by the p38 MAPK pathway, TLR4/NF-B pathway, and PI3K/Akt pathway [9599]

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K., & Wu, S

Although a single dedicated receptor has not been conclusively identified, experimental research indicates that BPC-157 influences several interconnected molecular systems, particularly nitric oxide signaling, angiogenic regulation, and focal adhesion associated pathways [1]