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The faster damaged tissue can stabilize, the less mechanical disruption occurs during the vulnerable early healing phase
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A., Hamimah, S., Arshad, A
MOTS-c modulates the FOXO transcription factors and regulates cellular senescence, the state in which aged or damaged cells stop dividing but refuse to die, instead secreting a cocktail of inflammatory signals known as the senescence-associated secretory phenotype (SASP)
Vascular calcification in chronic kidney disease: diversity in the vessel wall
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