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In rats, feeding activity and food intake decreased following intracerebroventricular injection of GLP-1, an effect counteracted by co-administration of a GLP-1 receptor antagonist (71, 72), suggesting central GLP-1 as a potent inhibitor of feeding

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However, because of their glucose-dependent mechanism of action, these agents tend to have a more significant effect on postprandial glucose than on fasting BG (40 mg/dL vs

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