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Rather than directly supplying NAD+ precursors (like NMN or NR), it works by blocking an enzyme that consumes nicotinamide, theoretically preserving the substrate pool for endogenous NAD+ production
Rarer ones include gallbladder problems, pancreatitis, and gastroparesis, a chronic stomach condition that can cause serious pain, vomiting, and malnutrition
Heres a quick recap of a few key distinctions between the drugs
Cagrilintide works through the amylin pathway, offering a different mechanism entirely
Your doctor can then decide whether you need to adjust your treatment plan
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