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Presented for research literature review only French JB et al
the biological complexity and potential for overlapping toxicities when designing combination therapies to exploit synergistic effects across pathways
Mechanistically, autoantibodies and interferon- present in the serum induce neutrophil ferroptosis through enhanced binding of the transcriptional repressor CREM to the glutathione peroxidase 4 ( Gpx4 , the key ferroptosis regulator) promoter, which leads to suppressed expression of Gpx4 and subsequent elevation of lipid-reactive oxygen species
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