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[1] Most of the time, those who are affected have no symptoms
GSH was shown to be the main inhibitor in the active site of the main protease (M pro ), the essential protein for virus invasion, and cysteine (Cys300) glutathionylation inhibits M pro activity by blocking its dimerization supporting the use of GSH in COVID-19 patients (Davis et al., 2021
This demonstrates the critical role of the CHAC2-NRF2 signaling pathway in protecting against oxidative stress and highlights CHAC2 as a potential therapeutic target for mitigating APAP-induced liver damage (Zhai et al., 2022)
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